Aesthetic Treatments for Keratosis Pilaris

Last updated: 9 August 2026

Keratosis pilaris: Understanding the "chicken skin" condition

Keratosis pilaris (KP), colloquially known as “chicken skin”, is an extremely common, benign skin condition characterised by small, rough, flesh-coloured or reddish bumps on the outer arms, thighs, cheeks, and buttocks. Affecting an estimated 50–80% of adolescents and 40% of adults, KP is the result of keratin protein plugging individual hair follicles. While medically harmless, the rough, bumpy texture can be a significant cosmetic concern for affected individuals.

Clinics offer evidence-based treatment protocols to effectively manage keratosis pilaris, improving skin texture and reducing the visible bumps and redness associated with this common condition.

The pathophysiology of keratosis pilaris

KP occurs when keratin, the tough protein that protects skin from infections and harmful substances, accumulates in the hair follicle opening, forming a hard plug. This process, known as follicular keratinisation, creates the characteristic small, rough bumps.

Types of keratosis pilaris

• KP albarough, colourless bumps without significant inflammation; the most common form
• KP rubrabumps surrounded by erythema (redness); more noticeable and often more distressing for patients
• KP rubra faceiireddish patches on the cheeks, often mistaken for rosacea or acne
• KP atrophicansa rarer form that can cause scarring, particularly on the face

Contributing Factors

• GeneticsKP runs in families and is linked to filaggrin gene mutations also associated with atopic dermatitis
• Atopic dermatitisKP is significantly more common in eczema patients
• Dry skinsymptoms worsen with dehydration and low humidity (typically worse in winter)
• Vitamin A deficiencythough rare in the UK, suboptimal vitamin A status may exacerbate KP
• Hormonal factorsKP often worsens during puberty and may improve with age

Professional treatment approaches

Chemical Exfoliation

Professional chemical peels are among the most effective treatments for KP:

• Salicylic acid peels (20–30%)lipophilic acid that penetrates into the follicle to dissolve keratin debris from within
• Lactic acid peelshydrating exfoliation particularly suitable for sensitive or dry KP-affected skin
• Combination peelsalternating between different acids can target multiple aspects of KP pathology

Microdermabrasion

Physical exfoliation using crystal or diamond-tip microdermabrasion can effectively remove the superficial keratin plugs, improving texture immediately. Regular sessions (every 2–4 weeks) maintain results. Best combined with topical treatment between sessions.

Laser and light therapies

• Pulsed dye laser (PDL)targets the vascular (red) component of KP rubra; effective for reducing erythema without addressing texture
• Fractional non-ablative laserstimulates collagen remodelling around affected follicles; emerging evidence for textural improvement
• IPLcan address redness component, particularly on facial KP rubra faceii
• Nd:YAG laserhas shown benefit for both the keratotic and erythematous components in darker skin types

Microneedling

Collagen induction therapy can improve KP texture by remodelling the skin around affected follicles. A 2022 pilot study showed significant improvement in KP severity scores after three microneedling sessions at 4-week intervals.

At-Home Management

Consistent home care is essential for maintaining professional treatment results:

• AHA/BHA lotionsproducts containing 10–15% glycolic acid, lactic acid, or 2% salicylic acid applied daily to affected areas
• Urea creams (10–20%)keratolytic and deeply hydrating; particularly effective for body KP
• Retinol body productsnormalise keratinisation with consistent use
• Gentle physical exfoliationa soft body brush or exfoliating cloth used 2–3 times weekly (avoid harsh scrubbing which can worsen inflammation)
• Ceramide-rich moisturisersapplied immediately after bathing to seal in hydration
• Avoid long, hot showersthese strip natural oils and worsen dryness

Treatment protocol: A suggested approach

1. Weeks 1–4: Establish daily AHA/BHA and moisturiser regimen; assess response
2. Weeks 4–12: Professional chemical peels or microdermabrasion every 2–3 weeks (4–6 sessions)
3. Weeks 12–16: Assess improvement; consider laser for residual redness if KP rubra component persists
4. Maintenance: Ongoing daily topical regimen; professional peel or microdermabrasion every 4–8 weeks as needed

Frequently asked questions

Will keratosis pilaris go away on its own?

KP often improves naturally with age, particularly after the age of 30. However, many adults continue to experience symptoms throughout life. Consistent management can keep symptoms well-controlled regardless of natural progression.

Is keratosis pilaris related to acne?

While KP and acne both involve follicular blockage, they are distinct conditions. KP results from keratin accumulation, while acne involves sebum, bacteria, and inflammation. Some treatments (retinoids, AHAs) are effective for both conditions.

Can diet affect keratosis pilaris?

Some anecdotal evidence suggests that eliminating dairy or gluten may improve KP in certain individuals, though rigorous clinical evidence is lacking. Ensuring adequate vitamin A and essential fatty acid intake may support skin health. Dietary changes should complement, not replace, evidence-based topical and professional treatments.

Is it safe to use exfoliating acids on keratosis pilaris during pregnancy?

Glycolic acid and lactic acid are generally considered safe during pregnancy. Salicylic acid should be used cautiously (small areas only) or avoided during pregnancy. Retinoid-based products should be discontinued during pregnancy. Always consult your healthcare provider.

This article is for informational purposes only and does not constitute medical advice. Always consult a qualified practitioner for personalised assessment and treatment. Individual results may vary.